According scientists at Newcastle University’s Institute for Ageing in the U.K. and Keio University School of Medicine, they say they have cracked the secret of why some people live a healthy and physically independent life over the age of 100: keeping inflammation down and telomeres long.
This month, the XPRIZE Foundation announced the winner of the Nokia Sensing XCHALLENGE. The global competition was aimed at accelerating the availability of hardware sensors and software sensing technology as a means to smarter digital health solutions. The winning device is called the Reusable Handheld Electrolyte and Lab Technology for Humans (rHEALTH) system. It can potentially run hundreds or even thousands of lab tests using a single drop of blood, and those tests, in turn, can be used to diagnose a range of diseases. (Video)
When people consume modern processed foods high in sugar, refined flour, and vegetable oils, they get sick.
The main reason why people all over the world are fatter and sicker than ever before is our modern diet. Everywhere modern processed foods go, chronic diseases like obesity, type 2 diabetes, and heart disease soon follow.
A normal mitochondria (left) contain distinctive folds known as cristae, but these folds are lost in damaged or dysfunctional mitochondria (right).
A team of Penn State scientists have discovered a “maternal age effect” that could be used to predict the accumulation of mitochondrial DNA mutations in maternal egg cells — and the transmission of these mutations to children — could provide valuable insights for genetic counseling. These mutations cause more than 200 diseases and contribute to others such as diabetes, cancer, Parkinson’s disease, and Alzheimer’s disease.
Loring (front row, center) with the Loring Lab Group at the Center for Regenerative Medicine.
Jeanne Loring, director of the Center for Regenerative Medicine at Scripps Research Institute, and her colleagues transplanted a set of cells into the spinal cords of mice that had lost use of their hind limbs to multiple sclerosis. Within a week, as the experimentalists had expected, the mice rejected the cells. But after another week, the mice began to walk.